Treatment with quercetin and 3′,4′-dihydroxyflavonol inhibits platelet function and reduces thrombus formation in vivo

Flavonoid intake and cardiovascular disease mortality in a prospective cohort of US adults

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Protein disulfide isomerase inhibitors constitute a new class of antithrombotic agents

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3′,4′-Dihydroxyflavonol reduces vascular contraction through Ca2+ desensitization in permeabilized rat mesenteric artery

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3′,4′-Dihydroxyflavonol Reduces Superoxide and Improves Nitric Oxide Function in Diabetic Rat Mesenteric Arteries

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Flavonols and cardiovascular disease

(2010) Francisco Perez-Vizcaino et al.   MOLECULAR ASPECTS OF MEDICINE

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(2010) Alan D. Michelson   NATURE REVIEWS DRUG DISCOVERY

Erythrocyte Hemolysis and Hemoglobin Oxidation Promote Ferric Chloride-induced Vascular Injury

(2009) Kevin J. Woollard et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Quercetin Glucuronide Inhibits Cell Migration and Proliferation by Platelet-Derived Growth Factor in Vascular Smooth Muscle Cells

(2009) Keisuke Ishizawa et al.   JOURNAL OF PHARMACOLOGICAL SCIENCES

A platelet tetraspanin superfamily member, CD151, is required for regulation of thrombus growth and stabilityin vivo

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The platelet release reaction: just when you thought platelet secretion was simple

(2008) Qiansheng Ren et al.   CURRENT OPINION IN HEMATOLOGY