Checkpoints in productive and latent infections with herpes simplex virus 1: conceptualization of the issues
出版年份 2011 全文链接
标题
Checkpoints in productive and latent infections with herpes simplex virus 1: conceptualization of the issues
作者
关键词
-
出版物
JOURNAL OF NEUROVIROLOGY
Volume 17, Issue 6, Pages 512-517
出版商
Springer Nature
发表日期
2011-11-04
DOI
10.1007/s13365-011-0058-x
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- The Checkpoints of Viral Gene Expression in Productive and Latent Infection: the Role of the HDAC/CoREST/LSD1/REST Repressor Complex
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- Disruption of HDAC/CoREST/REST repressor by dnREST reduces genome silencing and increases virulence of herpes simplex virus
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- Circadian CLOCK histone acetyl transferase localizes at ND10 nuclear bodies and enables herpes simplex virus gene expression
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- Analysis of Human Alphaherpesvirus MicroRNA Expression in Latently Infected Human Trigeminal Ganglia
- (2009) J. L. Umbach et al. JOURNAL OF VIROLOGY
- Inhibition of the histone demethylase LSD1 blocks α-herpesvirus lytic replication and reactivation from latency
- (2009) Yu Liang et al. NATURE MEDICINE
- De Novo Synthesis of VP16 Coordinates the Exit from HSV Latency In Vivo
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- The Two Functions of Herpes Simplex Virus 1 ICP0, Inhibition of Silencing by the CoREST/REST/HDAC Complex and Degradation of PML, Are Executed in Tandem
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- Chromatin control of herpes simplex virus lytic and latent infection
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- An acutely and latently expressed herpes simplex virus 2 viral microRNA inhibits expression of ICP34.5, a viral neurovirulence factor
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