Deletion of astroglial CXCL10 delays clinical onset but does not affect progressive axon loss in a murine autoimmune multiple sclerosis model

The Chemokine Receptor CXCR3 Is Degraded following Internalization and Is Replenished at the Cell Surface by De Novo Synthesis of Receptor

(2014) A. Meiser et al.   JOURNAL OF IMMUNOLOGY

Macrophages migrate in an activation-dependent manner to chemokines involved in neuroinflammation

(2014) Daphne YS Vogel et al.   Journal of Neuroinflammation

Th1-mediated experimental autoimmune encephalomyelitis is CXCR3 independent

(2013) Stephen J. Lalor et al.   EUROPEAN JOURNAL OF IMMUNOLOGY

Astrocytes regulate myelin clearance through recruitment of microglia during cuprizone-induced demyelination

(2012) Thomas Skripuletz et al.   BRAIN

IFN-gamma signaling in the central nervous system controls the course of experimental autoimmune encephalomyelitis independently of the localization and composition of inflammatory foci

(2012) Eunyoung Lee et al.   Journal of Neuroinflammation

CXCR3 ligands: redundant, collaborative and antagonistic functions

(2011) Joanna R Groom et al.   IMMUNOLOGY AND CELL BIOLOGY

CXCL10/CXCR3 signaling in glia cells differentially affects NMDA-induced cell death in CA and DG neurons of the mouse hippocampus

(2010) Hilmar R.J. van Weering et al.   HIPPOCAMPUS

A semiautomated targeted sampling method to assess optic nerve axonal loss in a rat model of glaucoma

(2010) Nephtali Marina et al.   Nature Protocols

An antibody to IP-10 is a potent antagonist of cell migrationin vitroandin vivoand does not affect disease in several animal models of inflammation

(2009) Fergus R. Byrne et al.   AUTOIMMUNITY

Initiation and Progression of Axonopathy in Experimental Autoimmune Encephalomyelitis

(2009) A. M. Soulika et al.   JOURNAL OF NEUROSCIENCE

Reactive Astrocytes Form Scar-Like Perivascular Barriers to Leukocytes during Adaptive Immune Inflammation of the CNS

(2009) R. R. Voskuhl et al.   JOURNAL OF NEUROSCIENCE

C-C chemokine receptor 6–regulated entry of TH-17 cells into the CNS through the choroid plexus is required for the initiation of EAE

(2009) Andrea Reboldi et al.   NATURE IMMUNOLOGY