Fractalkine (CX3CL1) enhances hippocampal N-methyl-d-aspartate receptor (NMDAR) function via d-serine and adenosine receptor type A2 (A2AR) activity
出版年份 2013 全文链接
标题
Fractalkine (CX3CL1) enhances hippocampal N-methyl-d-aspartate receptor (NMDAR) function via d-serine and adenosine receptor type A2 (A2AR) activity
作者
关键词
CX<sub>3</sub>CL1, NMDAR, Microglia, Hippocampus, <span class=EmphasisTypeSmallCaps >d, Adenosine receptors
出版物
Journal of Neuroinflammation
Volume 10, Issue 1, Pages -
出版商
Springer Nature
发表日期
2013-08-28
DOI
10.1186/1742-2094-10-108
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Activity of Adenosine Receptors Type 1 Is Required for CX3CL1-Mediated Neuroprotection and Neuromodulation in Hippocampal Neurons
- (2014) C. Lauro et al. JOURNAL OF IMMUNOLOGY
- Current status of chemokines in the adult CNS
- (2013) Annabelle Réaux-Le Goazigo et al. PROGRESS IN NEUROBIOLOGY
- Microglia, seen from the CX3CR1 angle
- (2013) Yochai Wolf et al. Frontiers in Cellular Neuroscience
- Synaptic and Extrasynaptic NMDA Receptors Are Gated by Different Endogenous Coagonists
- (2012) Thomas Papouin et al. CELL
- Therapeutic potential of the chemokine–receptor duo fractalkine/CX3CR1: an update
- (2012) Jan G D'Haese et al. EXPERT OPINION ON THERAPEUTIC TARGETS
- Cross-linking of serine racemase dimer by reactive oxygen species and reactive nitrogen species
- (2012) Wei Wang et al. JOURNAL OF NEUROSCIENCE RESEARCH
- d-Serine: The right or wrong isoform?
- (2011) Sabine A. Fuchs et al. BRAIN RESEARCH
- Adenosine A2A receptors control neuroinflammation and consequent hippocampal neuronal dysfunction
- (2011) Nelson Rebola et al. JOURNAL OF NEUROCHEMISTRY
- Modulation of GABA Transport by Adenosine A1R-A2AR Heteromers, Which Are Coupled to Both Gs- and Gi/o-Proteins
- (2011) S. Cristovao-Ferreira et al. JOURNAL OF NEUROSCIENCE
- CX3CL1 Is Neuroprotective in Permanent Focal Cerebral Ischemia in Rodents
- (2011) R. Cipriani et al. JOURNAL OF NEUROSCIENCE
- CX3CR1 deficiency alters hippocampal-dependent plasticity phenomena blunting the effects of enriched environment
- (2011) Laura Maggi Frontiers in Cellular Neuroscience
- Protein kinase C activity regulates d-serine availability in the brain
- (2010) Charles Vargas-Lopes et al. JOURNAL OF NEUROCHEMISTRY
- CX3CL1-induced modulation at CA1 synapses reveals multiple mechanisms of EPSC modulation involving adenosine receptor subtypes
- (2010) S. Piccinin et al. JOURNAL OF NEUROIMMUNOLOGY
- Minocycline Prevents Osmotic Demyelination Syndrome by Inhibiting the Activation of Microglia
- (2010) H. Suzuki et al. JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY
- Long-term potentiation depends on release of d-serine from astrocytes
- (2010) Christian Henneberger et al. NATURE
- Ex vivo cultures of microglia from young and aged rodent brain reveal age-related changes in microglial function
- (2010) eMalick G. Njie et al. NEUROBIOLOGY OF AGING
- Adenosine A1 Receptors and Microglial Cells Mediate CX3CL1-Induced Protection of Hippocampal Neurons Against Glu-Induced Death
- (2010) Clotilde Lauro et al. NEUROPSYCHOPHARMACOLOGY
- Tuning and Fine-Tuning of Synapses with Adenosine
- (2009) A. Sebastiao et al. Current Neuropharmacology
- LTP impairment by fractalkine/CX3CL1 in mouse hippocampus is mediated through the activity of adenosine receptor type 3 (A3R)
- (2009) Laura Maggi et al. JOURNAL OF NEUROIMMUNOLOGY
- Contribution of NMDA receptor-mediated component to the EPSP in mouse Schaffer collateral synapses under single pulse stimulation protocol
- (2008) Bogdan Neagu et al. BRAIN RESEARCH
- Neuronal viability is controlled by a functional relation between synaptic and extrasynaptic NMDA receptors
- (2008) F. Léveillé et al. FASEB JOURNAL
- Adenosine A2A Receptors Are Essential for Long-Term Potentiation of NMDA-EPSCs at Hippocampal Mossy Fiber Synapses
- (2008) Nelson Rebola et al. NEURON
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