RIP1 mediates the protection of geldanamycin on neuronal injury induced by oxygen-glucose deprivation combined with zVAD in primary cortical neurons

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A Signaling Cascade of Nuclear Calcium-CREB-ATF3 Activated by Synaptic NMDA Receptors Defines a Gene Repression Module That Protects against Extrasynaptic NMDA Receptor-Induced Neuronal Cell Death and Ischemic Brain Damage

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Geldanamycin and its analog induce cytotoxicity in cultured human retinal pigment epithelial cells

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Necrostatin Decreases Oxidative Damage, Inflammation, and Injury after Neonatal HI

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Characterisation of neuronal cell death in acute and delayed in vitro ischemia (oxygen–glucose deprivation) models

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Hsp90 cleavage by an oxidative stress leads to its client proteins degradation and cancer cell death

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TNF-α Induces Two Distinct Caspase-8 Activation Pathways

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Identification of a Molecular Signaling Network that Regulates a Cellular Necrotic Cell Death Pathway

(2008) Junichi Hitomi et al.   CELL

c-IAP1 and c-IAP2 Are Critical Mediators of Tumor Necrosis Factor α (TNFα)-induced NF-κB Activation

(2008) Eugene Varfolomeev et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Identification of RIP1 kinase as a specific cellular target of necrostatins

(2008) Alexei Degterev et al.   Nature Chemical Biology

Regulation of TNFR1 and CD95 signalling by receptor compartmentalization

(2008) Stefan Schütze et al.   NATURE REVIEWS MOLECULAR CELL BIOLOGY

Geldanamycin protects rat brain through overexpression of HSP70 and reducing brain edema after cerebral focal ischemia

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