Skeletal muscle Nur77 expression enhances oxidative metabolism and substrate utilization
出版年份 2012 全文链接
标题
Skeletal muscle Nur77 expression enhances oxidative metabolism and substrate utilization
作者
关键词
-
出版物
JOURNAL OF LIPID RESEARCH
Volume 53, Issue 12, Pages 2610-2619
出版商
American Society for Biochemistry & Molecular Biology (ASBMB)
发表日期
2012-10-02
DOI
10.1194/jlr.m029355
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- The Nuclear Receptor, Nor-1, Markedly Increases Type II Oxidative Muscle Fibers and Resistance to Fatigue
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- The common missense mutation D489N in TRIM32 causing limb girdle muscular dystrophy 2H leads to loss of the mutated protein in knock-in mice resulting in a Trim32-null phenotype
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- Insulin Resistance and Altered Systemic Glucose Metabolism in Mice Lacking Nur77
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- β-Adrenergic signaling regulates NR4A nuclear receptor and metabolic gene expression in multiple tissues☆
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- Mitochondrial Overload and Incomplete Fatty Acid Oxidation Contribute to Skeletal Muscle Insulin Resistance
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- The Orphan Nuclear Receptor, NOR-1, a Target of β-Adrenergic Signaling, Regulates Gene Expression that Controls Oxidative Metabolism in Skeletal Muscle
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- Mitochondrial biogenesis and healthy aging
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