期刊
JOURNAL OF LEUKOCYTE BIOLOGY
卷 84, 期 3, 页码 614-622出版社
WILEY
DOI: 10.1189/jlb.1107770
关键词
Tr1; Th3; CD8(+) Tregs; FoxP3; IL-10; TGF-beta; liver
资金
- NIAAA NIH HHS [R01 AA011576, R29 AA008577, AA011576, R21 AA016571, AA008577, R01 AA008577, AA016571] Funding Source: Medline
The mechanism behind the apparent lack of effective antiviral immune response in patients with chronic hepatitis C virus (HCV) infection is poorly understood. Although multiple levels of abnormalities have been identified in innate and adaptive immunity, it remains unclear if any of the subpopulations of T cells with regulatory capacity (Tregs) contribute to the induction and maintenance of HCV persistence. In this review, we summarize the current knowledge about Tregs as they relate to HCV infection.
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