Mutually exclusive redox forms of HMGB1 promote cell recruitment or proinflammatory cytokine release
出版年份 2012 全文链接
标题
Mutually exclusive redox forms of HMGB1 promote cell recruitment or proinflammatory cytokine release
作者
关键词
-
出版物
JOURNAL OF EXPERIMENTAL MEDICINE
Volume 209, Issue 9, Pages 1519-1528
出版商
Rockefeller University Press
发表日期
2012-08-07
DOI
10.1084/jem.20120189
参考文献
相关参考文献
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- HMGB1 promotes recruitment of inflammatory cells to damaged tissues by forming a complex with CXCL12 and signaling via CXCR4
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- Unknown
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- HMGB1 Is a Therapeutic Target for Sterile Inflammation and Infection
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- HMGB1 as an autophagy sensor in oxidative stress
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- Redox State-Dependent Interaction of HMGB1 and Cisplatin-Modified DNA
- (2011) Semi Park et al. BIOCHEMISTRY
- Therapeutic Potential of High Mobility Group Box-1 in Ischemic Injury and Tissue Regeneration
- (2011) Federico Biscetti et al. Current Vascular Pharmacology
- Kupffer Cells Hasten Resolution of Liver Immunopathology in Mouse Models of Viral Hepatitis
- (2011) Giovanni Sitia et al. PLoS Pathogens
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- Oxidation of the alarmin high-mobility group box 1 protein (HMGB1) during apoptosis
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- GFP-p65 knock-in mice as a tool to study NF-κB dynamics in vivo
- (2009) Rossana De Lorenzi et al. GENESIS
- DAMPs and inflammatory processes: the role of redox in the different outcomes
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- HMGB1 loves company
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- Sustained Oscillations of NF-κB Produce Distinct Genome Scanning and Gene Expression Profiles
- (2009) Myong-Hee Sung et al. PLoS One
- High-Mobility Group Box-1 Protein and Keratin-18, Circulating Serum Proteins Informative of Acetaminophen-Induced Necrosis and Apoptosis In Vivo
- (2009) Daniel J. Antoine et al. TOXICOLOGICAL SCIENCES
- High-Mobility Group Box-1 in Ischemia-Reperfusion Injury of the Heart
- (2008) Martin Andrassy et al. CIRCULATION
- Induction of Immunological Tolerance by Apoptotic Cells Requires Caspase-Dependent Oxidation of High-Mobility Group Box-1 Protein
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- The HMGB1 Receptor RAGE Mediates Ischemic Brain Damage
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