B cell–intrinsic signaling through IL-21 receptor and STAT3 is required for establishing long-lived antibody responses in humans
出版年份 2010 全文链接
标题
B cell–intrinsic signaling through IL-21 receptor and STAT3 is required for establishing long-lived antibody responses in humans
作者
关键词
-
出版物
JOURNAL OF EXPERIMENTAL MEDICINE
Volume 207, Issue 1, Pages 155-171
出版商
Rockefeller University Press
发表日期
2010-01-05
DOI
10.1084/jem.20091706
参考文献
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- IL-21-Induced Isotype Switching to IgG and IgA by Human Naive B Cells Is Differentially Regulated by IL-4
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- Viral Double-Stranded RNA Triggers Ig Class Switching by Activating Upper Respiratory Mucosa B Cells through an Innate TLR3 Pathway Involving BAFF
- (2014) W. Xu et al. JOURNAL OF IMMUNOLOGY
- Memory B cells: Effectors of long-lived immune responses
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- Molecular explanation for the contradiction between systemic Th17 defect and localized bacterial infection in hyper-IgE syndrome
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- Generation of T Follicular Helper Cells Is Mediated by Interleukin-21 but Independent of T Helper 1, 2, or 17 Cell Lineages
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- Novel signal transducer and activator of transcription 3 (STAT3) mutations, reduced TH17 cell numbers, and variably defective STAT3 phosphorylation in hyper-IgE syndrome
- (2008) Ellen D. Renner et al. JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY
- Deficiency of Th17 cells in hyper IgE syndrome due to mutations inSTAT3
- (2008) Cindy S. Ma et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Mutations inSTAT3andIL12RB1impair the development of human IL-17–producing T cells
- (2008) Ludovic de Beaucoudrey et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Impaired TH17 cell differentiation in subjects with autosomal dominant hyper-IgE syndrome
- (2008) Joshua D. Milner et al. NATURE
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