Nucleotide excision repair efficiency in quiescent human fibroblasts is modulated by circadian clock
出版年份 2015 全文链接
标题
Nucleotide excision repair efficiency in quiescent human fibroblasts is modulated by circadian clock
作者
关键词
-
出版物
NUCLEIC ACIDS RESEARCH
Volume 43, Issue 4, Pages 2126-2137
出版商
Oxford University Press (OUP)
发表日期
2015-02-07
DOI
10.1093/nar/gkv081
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- DNA Repair Synthesis and Ligation Affect the Processing of Excised Oligonucleotides Generated by Human Nucleotide Excision Repair
- (2014) Michael G. Kemp et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Modulation of ATR-mediated DNA damage checkpoint response by cryptochrome 1
- (2014) T.-H. Kang et al. NUCLEIC ACIDS RESEARCH
- NER initiation factors, DDB2 and XPC, regulate UV radiation response by recruiting ATR and ATM kinases to DNA damage sites
- (2013) Alo Ray et al. DNA REPAIR
- UV-Induced Nuclear Import of XPA Is Mediated by Importin-α4 in An ATR-Dependent Manner
- (2013) Zhengke Li et al. PLoS One
- The Efficiency of Homologous Recombination and Non-Homologous End Joining Systems in Repairing Double-Strand Breaks during Cell Cycle Progression
- (2013) Leonardo Bee et al. PLoS One
- The deoxynucleotide triphosphohydrolase SAMHD1 is a major regulator of DNA precursor pools in mammalian cells
- (2013) E. Franzolin et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Cell-autonomous circadian DNA damage response
- (2012) Roman V. Kondratov CELL CYCLE
- Effect of circadian clock mutations on DNA damage response in mammalian cells
- (2012) Shobhan Gaddameedhi et al. CELL CYCLE
- Establishment of a Microplate-Formatted Cell-Based Immunoassay for Rapid Analysis of Nucleotide Excision Repair Ability in Human Primary Cells
- (2012) Mari Nishinaga et al. PHOTOCHEMISTRY AND PHOTOBIOLOGY
- Ordered changes in histone modifications at the core of the Arabidopsis circadian clock
- (2012) J. Malapeira et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Mammalian ribonucleotide reductase subunit p53R2 is required for mitochondrial DNA replication and DNA repair in quiescent cells
- (2012) G. Pontarin et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Brain and muscle Arnt-like protein-1 (BMAL1) controls circadian cell proliferation and susceptibility to UVB-induced DNA damage in the epidermis
- (2012) M. Geyfman et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- NONO couples the circadian clock to the cell cycle
- (2012) E. Kowalska et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Transcriptional Architecture and Chromatin Landscape of the Core Circadian Clock in Mammals
- (2012) N. Koike et al. SCIENCE
- Cytometric detection of chromatin relaxation, an early reporter of DNA damage response
- (2011) H. Dorota Halicka et al. CELL CYCLE
- Tip60: Connecting chromatin to DNA damage signaling
- (2011) Yingli Sun et al. CELL CYCLE
- DNA damage response and transcription
- (2011) Saskia Lagerwerf et al. DNA REPAIR
- DNA Damage-Induced RORα Is Crucial for p53 Stabilization and Increased Apoptosis
- (2011) Hyunkyung Kim et al. MOLECULAR CELL
- γH2Ax: Biomarker of Damage or Functional Participant in DNA Repair “All that Glitters Is not Gold!”
- (2011) James E. Cleaver PHOTOCHEMISTRY AND PHOTOBIOLOGY
- XPA-Mediated Regulation of Global Nucleotide Excision Repair by ATR Is p53-Dependent and Occurs Primarily in S-Phase
- (2011) Zhengke Li et al. PLoS One
- Control of skin cancer by the circadian rhythm
- (2011) S. Gaddameedhi et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Circadian rhythms and cancer
- (2010) Sigal Gery et al. CELL CYCLE
- Open, repair and close again: Chromatin dynamics and the response to UV-induced DNA damage
- (2010) Zoraya Palomera-Sanchez et al. DNA REPAIR
- Circadian clock control of the cellular response to DNA damage
- (2010) Aziz Sancar et al. FEBS LETTERS
- Per3, a circadian gene, is required for Chk2 activation in human cells
- (2010) Jun-Sub Im et al. FEBS LETTERS
- The mammalian clock component PERIOD2 coordinates circadian output by interaction with nuclear receptors
- (2010) I. Schmutz et al. GENES & DEVELOPMENT
- Drosophilap53 Is Required to Increase the Levels of the dKDM4B Demethylase after UV-induced DNA Damage to Demethylate Histone H3 Lysine 9
- (2010) Zoraya Palomera-Sanchez et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Regulation of nucleotide excision repair activity by transcriptional and post-transcriptional control of the XPA protein
- (2010) T.-H. Kang et al. NUCLEIC ACIDS RESEARCH
- Circadian control of XPA and excision repair of cisplatin-DNA damage by cryptochrome and HERC2 ubiquitin ligase
- (2010) T.-H. Kang et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Screen for DNA-damage-responsive histone modifications identifies H3K9Ac and H3K56Ac in human cells
- (2009) Jorrit V Tjeertes et al. EMBO JOURNAL
- Circadian oscillation of nucleotide excision repair in mammalian brain
- (2009) Tae-Hong Kang et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Harmonics of Circadian Gene Transcription in Mammals
- (2009) Michael E. Hughes et al. PLoS Genetics
- The NAD+-Dependent Deacetylase SIRT1 Modulates CLOCK-Mediated Chromatin Remodeling and Circadian Control
- (2008) Yasukazu Nakahata et al. CELL
- IL-12 Deficiency Exacerbates Inflammatory Responses in UV-Irradiated Skin and Skin Tumors
- (2008) Syed M. Meeran et al. JOURNAL OF INVESTIGATIVE DERMATOLOGY
- Ribonucleotide reduction is a cytosolic process in mammalian cells independently of DNA damage
- (2008) G. Pontarin et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
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