TMEM16E (GDD1) Exhibits Protein Instability and Distinct Characteristics in Chloride Channel/Pore Forming Ability
出版年份 2013 全文链接
标题
TMEM16E (GDD1) Exhibits Protein Instability and Distinct Characteristics in Chloride Channel/Pore Forming Ability
作者
关键词
-
出版物
JOURNAL OF CELLULAR PHYSIOLOGY
Volume 229, Issue 2, Pages 181-190
出版商
Wiley
发表日期
2013-07-11
DOI
10.1002/jcp.24431
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- TMEM16F is a component of a Ca2+-activated Cl− channel but not a volume-sensitive outwardly rectifying Cl− channel
- (2013) Takahiro Shimizu et al. AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
- TMEM16F (Anoctamin 6), an anion channel of delayed Ca2+activation
- (2013) Søren Grubb et al. JOURNAL OF GENERAL PHYSIOLOGY
- TMEM16F Forms a Ca2+-Activated Cation Channel Required for Lipid Scrambling in Platelets during Blood Coagulation
- (2012) Huanghe Yang et al. CELL
- Explaining Calcium-Dependent Gating of Anoctamin-1 Chloride Channels Requires a Revised Topology
- (2012) Kuai Yu et al. CIRCULATION RESEARCH
- The voltage dependence of the TMEM16B/anoctamin2 calcium-activated chloride channel is modified by mutations in the first putative intracellular loop
- (2012) Valentina Cenedese et al. JOURNAL OF GENERAL PHYSIOLOGY
- Anoctamin 6 is an essential component of the outwardly rectifying chloride channel
- (2011) J. R. Martins et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Voltage- and calcium-dependent gating of TMEM16A/Ano1 chloride channels are physically coupled by the first intracellular loop
- (2011) Q. Xiao et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Recessive Mutations in the Putative Calcium-Activated Chloride Channel Anoctamin 5 Cause Proximal LGMD2L and Distal MMD3 Muscular Dystrophies
- (2010) Véronique Bolduc et al. AMERICAN JOURNAL OF HUMAN GENETICS
- A founder mutation in Anoctamin 5 is a major cause of limb girdle muscular dystrophy
- (2010) D. Hicks et al. BRAIN
- Angiotensin I-Converting Enzyme Gln1069Arg Mutation Impairs Trafficking to the Cell Surface Resulting in Selective Denaturation of the C-Domain
- (2010) Sergei M. Danilov et al. PLoS One
- Regulation of TMEM16A Chloride Channel Properties by Alternative Splicing
- (2009) Loretta Ferrera et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- TMEM16B, A Novel Protein with Calcium-Dependent Chloride Channel Activity, Associates with a Presynaptic Protein Complex in Photoreceptor Terminals
- (2009) H. Stohr et al. JOURNAL OF NEUROSCIENCE
- TMEM16B induces chloride currents activated by calcium in mammalian cells
- (2009) Simone Pifferi et al. PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY
- ANO2 is the cilial calcium-activated chloride channel that may mediate olfactory amplification
- (2009) Aaron B. Stephan et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Involvement of cytoskeletal integrity in the regulation of Cl- and amylase secretion from rat parotid acinar cells
- (2008) Hiroyasu KONGO et al. BIOMEDICAL RESEARCH-TOKYO
- Expression Cloning of TMEM16A as a Calcium-Activated Chloride Channel Subunit
- (2008) Björn Christian Schroeder et al. CELL
- Anoctamin/TMEM16 family members are Ca2+-activated Cl−channels
- (2008) H. Criss Hartzell et al. JOURNAL OF PHYSIOLOGY-LONDON
- TMEM16A confers receptor-activated calcium-dependent chloride conductance
- (2008) Young Duk Yang et al. NATURE
- TMEM16A, A Membrane Protein Associated with Calcium-Dependent Chloride Channel Activity
- (2008) A. Caputo et al. SCIENCE
Add your recorded webinar
Do you already have a recorded webinar? Grow your audience and get more views by easily listing your recording on Peeref.
Upload NowBecome a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get Started