ClC-3 is a main component of background chloride channels activated under isotonic conditions by autocrine ATP in nasopharyngeal carcinoma cells

ClC-2 Voltage-Gated Channels Constitute Part of the Background Conductance and Assist Chloride Extrusion

(2010) I. Rinke et al.   JOURNAL OF NEUROSCIENCE

Functional and molecular characterizations of chloride channels in rat pleural mesothelial cells

(2009) Yuya Yoshise et al.   EUROPEAN JOURNAL OF PHARMACOLOGY

Cardiac-specific, inducible ClC-3 gene deletion eliminates native volume-sensitive chloride channels and produces myocardial hypertrophy in adult mice

(2009) Dazhi Xiong et al.   JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY

ATP release from non-excitable cells

(2009) Helle A. Praetorius et al.   Purinergic Signalling

ATP as a mediator of macula densa cell signalling

(2009) P. Darwin Bell et al.   Purinergic Signalling

Ectonucleotidases in the kidney

(2009) David G. Shirley et al.   Purinergic Signalling

Suppression of ClC-3 channel expression reduces migration of nasopharyngeal carcinoma cells

(2008) Jianwen Mao et al.   BIOCHEMICAL PHARMACOLOGY

Maxi-anion channel as a candidate pathway for osmosensitive ATP release from mouse astrocytes in primary culture

(2008) Hong-Tao Liu et al.   CELL RESEARCH

Development of selective agonists and antagonists of P2Y receptors

(2008) Kenneth A. Jacobson et al.   Purinergic Signalling

Spatial Distribution of Maxi-Anion Channel on Cardiomyocytes Detected by Smart-Patch Technique

(2007) Amal K. Dutta et al.   BIOPHYSICAL JOURNAL