P2X7 purinoceptor alterations in dystrophic mdx mouse muscles: relationship to pathology and potential target for treatment
出版年份 2011 全文链接
标题
P2X7 purinoceptor alterations in dystrophic mdx mouse muscles: relationship to pathology and potential target for treatment
作者
关键词
-
出版物
JOURNAL OF CELLULAR AND MOLECULAR MEDICINE
Volume 16, Issue 5, Pages 1026-1037
出版商
Wiley
发表日期
2011-07-28
DOI
10.1111/j.1582-4934.2011.01397.x
参考文献
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Extracellular ATP signaling during differentiation of C2C12 skeletal muscle cells: role in proliferation
- (2011) Tiziana Martinello et al. MOLECULAR AND CELLULAR BIOCHEMISTRY
- Inflammasome Up-Regulation and Activation in Dysferlin-Deficient Skeletal Muscle
- (2010) Rashmi Rawat et al. AMERICAN JOURNAL OF PATHOLOGY
- Pannexin-I/P2X 7 Purinergic Receptor Channels Mediate the Release of Cardioprotectants Induced by Ischemic Pre- and Postconditioning
- (2010) Donald A. Vessey et al. JOURNAL OF CARDIOVASCULAR PHARMACOLOGY AND THERAPEUTICS
- Prevention of muscle fibrosis and myonecrosis inmdxmice by suramin, a TGF-β1 blocker
- (2010) Ana Paula Tiemi Taniguti et al. MUSCLE & NERVE
- Mutation in dystrophin-encoding gene affects energy metabolism in mouse myoblasts
- (2009) Marta Onopiuk et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Mammalian P2X7 receptor pharmacology: comparison of recombinant mouse, rat and human P2X7 receptors
- (2009) Diana L Donnelly-Roberts et al. BRITISH JOURNAL OF PHARMACOLOGY
- Muscular dystrophy begins early in embryonic development deriving from stem cell loss and disrupted skeletal muscle formation
- (2009) D. Merrick et al. Disease Models & Mechanisms
- The P2X7 purinergic receptor: from physiology to neurological disorders
- (2009) Stephen D. Skaper et al. FASEB JOURNAL
- Changes in the expression of insulin-like growth factor 1 variants in the postnatal brain development and in neonatal hypoxia–ischaemia
- (2009) Małgorzata Beręsewicz et al. INTERNATIONAL JOURNAL OF DEVELOPMENTAL NEUROSCIENCE
- A Functional P2X7 Splice Variant with an Alternative Transmembrane Domain 1 Escapes Gene Inactivation in P2X7 Knock-out Mice
- (2009) Annette Nicke et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Analysis of Assembly and Trafficking of Native P2X4 and P2X7 Receptor Complexes in Rodent Immune Cells
- (2009) Miyyada Boumechache et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- ATP Released by Electrical Stimuli Elicits Calcium Transients and Gene Expression in Skeletal Muscle
- (2009) Sonja Buvinic et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Differential Regulation of P2X7 Receptor Activation by Extracellular Nicotinamide Adenine Dinucleotide and Ecto-ADP-Ribosyltransferases in Murine Macrophages and T Cells
- (2009) S. Hong et al. JOURNAL OF IMMUNOLOGY
- Diltiazem and verapamil protect dystrophin-deficient muscle fibers ofMDXmice from degeneration: A potential role in calcium buffering and sarcolemmal stability
- (2009) Cintia Yuri Matsumura et al. MUSCLE & NERVE
- Calcium influx is sufficient to induce muscular dystrophy through a TRPC-dependent mechanism
- (2009) Douglas P. Millay et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- P2X7: a growth-promoting receptor—implications for cancer
- (2009) Francesco Di Virgilio et al. Purinergic Signalling
- Signaling at Purinergic P2X Receptors
- (2008) Annmarie Surprenant et al. Annual Review of Physiology
- Interleukin-6 Is an Essential Regulator of Satellite Cell-Mediated Skeletal Muscle Hypertrophy
- (2008) Antonio L. Serrano et al. Cell Metabolism
- ATP-induced P2X7-associated uptake of large molecules involves distinct mechanisms for cations and anions in macrophages
- (2008) J. Schachter et al. JOURNAL OF CELL SCIENCE
- The P2X7Receptor Channel Pore Dilates under Physiological Ion Conditions
- (2008) Zonghe Yan et al. JOURNAL OF GENERAL PHYSIOLOGY
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