Mastl is required for timely activation of APC/C in meiosis I and Cdk1 reactivation in meiosis II
出版年份 2014 全文链接
标题
Mastl is required for timely activation of APC/C in meiosis I and Cdk1 reactivation in meiosis II
作者
关键词
-
出版物
JOURNAL OF CELL BIOLOGY
Volume 206, Issue 7, Pages 843-853
出版商
Rockefeller University Press
发表日期
2014-09-23
DOI
10.1083/jcb.201406033
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- PP2A delays APC/C-dependent degradation of separase-associated but not free securin
- (2014) S. Hellmuth et al. EMBO JOURNAL
- Cyclin B–Cdk1 inhibits protein phosphatase PP2A-B55 via a Greatwall kinase–independent mechanism
- (2014) Eiichi Okumura et al. JOURNAL OF CELL BIOLOGY
- Dual-mode regulation of the APC/C by CDK1 and MAPK controls meiosis I progression and fidelity
- (2014) Ibtissem Nabti et al. JOURNAL OF CELL BIOLOGY
- Phosphorylation of ARPP19 by protein kinase A prevents meiosis resumption in Xenopus oocytes
- (2014) Aude Dupré et al. Nature Communications
- Greatwall Kinase Is Required for Meiotic Maturation in Porcine Oocytes1
- (2013) Ying-Hua Li et al. BIOLOGY OF REPRODUCTION
- Inhibition of Protein Phosphatase 2A Radiosensitizes Pancreatic Cancers by Modulating CDC25C/CDK1 and Homologous Recombination Repair
- (2013) D. Wei et al. CLINICAL CANCER RESEARCH
- Cell cycle regulation of Greatwall kinase nuclear localization facilitates mitotic progression
- (2013) Peng Wang et al. JOURNAL OF CELL BIOLOGY
- Increased CDK1 activity determines the timing of kinetochore-microtubule attachments in meiosis I
- (2013) Olga Davydenko et al. JOURNAL OF CELL BIOLOGY
- The phosphorylation of ARPP19 by Greatwall renders the auto-amplification of MPF independently of PKA in Xenopus oocytes
- (2013) A. Dupre et al. JOURNAL OF CELL SCIENCE
- The BEG (PP2A-B55/ENSA/Greatwall) Pathway Ensures Cytokinesis follows Chromosome Separation
- (2013) Michael J. Cundell et al. MOLECULAR CELL
- DIM (3,3'-diindolylmethane) confers protection against ionizing radiation by a unique mechanism
- (2013) S. Fan et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Greatwall is essential to prevent mitotic collapse after nuclear envelope breakdown in mammals
- (2013) M. Alvarez-Fernandez et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Protein phosphatases and their regulation in the control of mitosis
- (2012) Satoru Mochida et al. EMBO REPORTS
- Bypassing the Greatwall-Endosulfine Pathway: Plasticity of a Pivotal Cell-Cycle Regulatory Module in Drosophila melanogaster and Caenorhabditis elegans
- (2012) M.-Y. Kim et al. GENETICS
- Cyclin-dependent kinase 1 (Cdk1) is essential for cell division and suppression of DNA re-replication but not for liver regeneration
- (2012) M. K. Diril et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Suppression of PP2A is critical for protection of melanoma cells upon endoplasmic reticulum stress
- (2012) K H Tay et al. Cell Death & Disease
- The overlooked greatwall: a new perspective on mitotic control
- (2012) D. M. Glover Open Biology
- Complete Kinetochore Tracking Reveals Error-Prone Homologous Chromosome Biorientation in Mammalian Oocytes
- (2011) Tomoya S. Kitajima et al. CELL
- Essential Role of Protein Phosphatase 2A in Metaphase II Arrest and Activation of Mouse Eggs Shown by Okadaic Acid, Dominant Negative Protein Phosphatase 2A, and FTY720
- (2011) Heng-Yu Chang et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Switches and latches: a biochemical tug-of-war between the kinases and phosphatases that control mitosis
- (2011) M. R. Domingo-Sananes et al. PHILOSOPHICAL TRANSACTIONS OF THE ROYAL SOCIETY B-BIOLOGICAL SCIENCES
- Suppression of Scant Identifies Endos as a Substrate of Greatwall Kinase and a Negative Regulator of Protein Phosphatase 2A in Mitosis
- (2011) Hélène Rangone et al. PLoS Genetics
- PP2A-Twins Is Antagonized by Greatwall and Collaborates with Polo for Cell Cycle Progression and Centrosome Attachment to Nuclei in Drosophila Embryos
- (2011) Peng Wang et al. PLoS Genetics
- MASTL is the human ortholog of Greatwall kinase that facilitates mitotic entry, anaphase and cytokinesis
- (2010) Erik Voets et al. CELL CYCLE
- Loss of human Greatwall results in G2 arrest and multiple mitotic defects due to deregulation of the cyclin B-Cdc2/PP2A balance
- (2010) Andrew Burgess et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Enforcing the Greatwall in Mitosis
- (2010) D. M. Virshup et al. SCIENCE
- The Substrate of Greatwall Kinase, Arpp19, Controls Mitosis by Inhibiting Protein Phosphatase 2A
- (2010) A. Gharbi-Ayachi et al. SCIENCE
- Greatwall Phosphorylates an Inhibitor of Protein Phosphatase 2A That Is Essential for Mitosis
- (2010) S. Mochida et al. SCIENCE
- Cdc20 Is Critical for Meiosis I and Fertility of Female Mice
- (2010) Fang Jin et al. PLoS Genetics
- CDC14B Acts Through FZR1 (CDH1) to Prevent Meiotic Maturation of Mouse Oocytes1
- (2009) Karen Schindler et al. BIOLOGY OF REPRODUCTION
- Regulation of APC/C Activity in Oocytes by a Bub1-Dependent Spindle Assembly Checkpoint
- (2009) Barry E. McGuinness et al. CURRENT BIOLOGY
- Greatwall maintains mitosis through regulation of PP2A
- (2009) Suzanne Vigneron et al. EMBO JOURNAL
- Regulated activity of PP2A–B55δ is crucial for controlling entry into and exit from mitosis in Xenopus egg extracts
- (2009) Satoru Mochida et al. EMBO JOURNAL
- Regulation of APC/C Activators in Mitosis and Meiosis
- (2008) Jillian A. Pesin et al. Annual Review of Cell and Developmental Biology
- -Endosulfine is a conserved protein required for oocyte meiotic maturation in Drosophila
- (2008) J. R. Von Stetina et al. DEVELOPMENT
- Regulation of Mih1/Cdc25 by protein phosphatase 2A and casein kinase 1
- (2008) Gayatri Pal et al. JOURNAL OF CELL BIOLOGY
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