A new humanized ataxin-3 knock-in mouse model combines the genetic features, pathogenesis of neurons and glia and late disease onset of SCA3/MJD

标题
A new humanized ataxin-3 knock-in mouse model combines the genetic features, pathogenesis of neurons and glia and late disease onset of SCA3/MJD
作者
关键词
Ataxin-3, Mouse, Knock-in, Knockin, SCA3, MJD, Ataxia, Spinocerebellar, CAG repeats, Serpina3n, Polyglutamine
出版物
NEUROBIOLOGY OF DISEASE
Volume 73, Issue -, Pages 174-188
出版商
Elsevier BV
发表日期
2014-10-09
DOI
10.1016/j.nbd.2014.09.020

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