标题
Inherited Pain
作者
关键词
-
出版物
JOURNAL OF BIOLOGICAL CHEMISTRY
Volume 289, Issue 4, Pages 1971-1980
出版商
American Society for Biochemistry & Molecular Biology (ASBMB)
发表日期
2013-12-06
DOI
10.1074/jbc.m113.502211
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Two Alternative Conformations of a Voltage-Gated Sodium Channel
- (2013) Ching-Ju Tsai et al. JOURNAL OF MOLECULAR BIOLOGY
- A Novel SCN9A Mutation Responsible for Primary Erythromelalgia and Is Resistant to the Treatment of Sodium Channel Blockers
- (2013) Min-Tzu Wu et al. PLoS One
- Small nerve fibres, small hands and small feet: a new syndrome of pain, dysautonomia and acromesomelia in a kindred with a novel NaV1.7 mutation
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- Deletion mutation of sodium channel NaV1.7 in inherited erythromelalgia: enhanced slow inactivation modulates dorsal root ganglion neuron hyperexcitability
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- Inhibition of Nav 4 Peptide-Mediated Resurgent Sodium Currents in Nav1.7 Channels by Carbamazepine, Riluzole, and Anandamide
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- The crystal structure of a voltage-gated sodium channel
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- Alternative splicing may contribute to time-dependent manifestation of inherited erythromelalgia
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- A new Nav1.7 sodium channel mutation I234T in a child with severe pain
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- Human voltage-gated sodium channel mutations that cause inherited neuronal and muscle channelopathies increase resurgent sodium currents
- (2010) Brian W. Jarecki et al. JOURNAL OF CLINICAL INVESTIGATION
- Nav1.7 mutations associated with paroxysmal extreme pain disorder, but not erythromelalgia, enhance Navβ4 peptide-mediated resurgent sodium currents
- (2010) Jonathan W. Theile et al. JOURNAL OF PHYSIOLOGY-LONDON
- Sodium channelopathies and pain
- (2010) Angelika Lampert et al. PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY
- Voltage-clamp and current-clamp recordings from mammalian DRG neurons
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- NaV1.7 Gain-of-Function Mutations as a Continuum: A1632E Displays Physiological Changes Associated with Erythromelalgia and Paroxysmal Extreme Pain Disorder Mutations and Produces Symptoms of Both Disorders
- (2008) M. Estacion et al. JOURNAL OF NEUROSCIENCE
- Paroxysmal extreme pain disorder mutations within the D3/S4-S5 linker of Nav1.7 cause moderate destabilization of fast inactivation
- (2008) Brian W. Jarecki et al. JOURNAL OF PHYSIOLOGY-LONDON
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