Dioxin Silences Gonadotropin Expression in Perinatal Pups by Inducing Histone Deacetylases

2,3,7,8-Tetrachlorodibenzo-p-dioxin potentially attenuates the gene expression of pituitary gonadotropin β-subunits in a fetal age-specific fashion: a comparative study using cultured pituitaries

(2011) Tomoki Takeda et al.   JOURNAL OF TOXICOLOGICAL SCIENCES

Female reproductive maturation in the absence of kisspeptin/GPR54 signaling

(2011) Christian Mayer et al.   NATURE NEUROSCIENCE

Mechanisms of FSH synthesis: what we know, what we don't, and why you should care

(2010) Daniel J. Bernard et al.   FERTILITY AND STERILITY

Activin A induction of FSHβ subunit transcription requires SMAD4 in immortalized gonadotropes

(2010) Ying Wang et al.   JOURNAL OF MOLECULAR ENDOCRINOLOGY

Maternal exposure to dioxin reduces hypothalamic but not pituitary metabolome in fetal rats: a possible mechanism for a fetus-specific reduction in steroidogenesis

(2010) Yuki Matsumoto et al.   JOURNAL OF TOXICOLOGICAL SCIENCES

Embryonic gonadotropin-releasing hormone signaling is necessary for maturation of the male reproductive axis

(2010) S. Wen et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Maternal Exposure to Dioxin Disrupts Gonadotropin Production in Fetal Rats and Imprints Defects in Sexual Behavior

(2009) T. Takeda et al.   JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS

Regulation of the steroidogenic acute regulatory protein gene expression: present and future perspectives

(2009) P. R. Manna et al.   MOLECULAR HUMAN REPRODUCTION

Estradiol and the Developing Brain

(2008) MARGARET M. McCARTHY   PHYSIOLOGICAL REVIEWS

Histone deacetylases and repression of the gonadotropin genes

(2008) Philippa Melamed   TRENDS IN ENDOCRINOLOGY AND METABOLISM