Specificity of the dRP/AP Lyase of Ku Promotes Nonhomologous End Joining (NHEJ) Fidelity at Damaged Ends

Ku is a 5′-dRP/AP lyase that excises nucleotide damage near broken ends

(2010) Steven A. Roberts et al.   NATURE

Base Damage Immediately Upstream from Double-Strand Break Ends is a More Severe Impediment to Nonhomologous End Joining than Blocked 3′-Termini

(2010) Kamal Datta et al.   RADIATION RESEARCH

Ku Regulates the Non-Homologous End Joining Pathway Choice of DNA Double-Strand Break Repair in Human Somatic Cells

(2010) Farjana Fattah et al.   PLoS Genetics

Apex1 can cleave complex clustered DNA lesions in cells

(2009) Svitlana Malyarchuk et al.   DNA REPAIR

Ku antigen interacts with abasic sites

(2008) Ekaterina S. Ilina et al.   BIOCHIMICA ET BIOPHYSICA ACTA-PROTEINS AND PROTEOMICS

Interplay of two major repair pathways in the processing of complex double-strand DNA breaks

(2008) Tracey A. Dobbs et al.   DNA REPAIR

Single-strand break repair and genetic disease

(2008) Keith W. Caldecott   NATURE REVIEWS GENETICS

DNA repair of clustered lesions in mammalian cells: involvement of non-homologous end-joining

(2008) S. Malyarchuk et al.   NUCLEIC ACIDS RESEARCH

Apn1 and Apn2 endonucleases prevent accumulation of repair-associated DNA breaks in budding yeast as revealed by direct chromosomal analysis

(2008) Wenjian Ma et al.   NUCLEIC ACIDS RESEARCH