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CXCL12-induced glioblastoma cell migration requires intermediate conductance Ca2+-activated K+ channel activity

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3-Bromopyruvate induces necrotic cell death in sensitive melanoma cell lines

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H2O2 disposal in cardiolipin-enriched brain mitochondria is due to increased cytochrome c peroxidase activity

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3-Bromopyruvate: A New Targeted Antiglycolytic Agent and a Promise for Cancer Therapy

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Glucose Deprivation Contributes to the Development of KRAS Pathway Mutations in Tumor Cells

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Inhibition of energy-producing pathways of HepG2 cells by 3-bromopyruvate1

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Cardiolipin deficiency leads to decreased cardiolipin peroxidation and increased resistance of cells to apoptosis

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Role of reactive oxygen species-mediated mitochondrial dysregulation in 3-bromopyruvate induced cell death in hepatoma cells

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Loss of cardiolipin in palmitate-treated GL15 glioblastoma cells favors cytochrome c release from mitochondria leading to apoptosis

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