Monascin and AITC Attenuate Methylglyoxal-Induced PPARγ Phosphorylation and Degradation through Inhibition of the Oxidative Stress/PKC Pathway Depending on Nrf2 Activation
Monascin and AITC Attenuate Methylglyoxal-Induced PPARγ Phosphorylation and Degradation through Inhibition of the Oxidative Stress/PKC Pathway Depending on Nrf2 Activation
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