Activation of lytic cycle of Epstein-Barr virus by suberoylanilide hydroxamic acid leads to apoptosis and tumor growth suppression of nasopharyngeal carcinoma

Epstein–Barr Virus (EBV) Rta-Mediated EBV and Kaposi's Sarcoma-Associated Herpesvirus Lytic Reactivations in 293 Cells

(2011) Yen-Ju Chen et al.   PLoS One

The interplay between Epstein-Barr virus and the immune system: a rationale for adoptive cell therapy of EBV-related disorders

(2010) A. Merlo et al.   HAEMATOLOGICA

STAT3 activation contributes directly to Epstein-Barr virus-mediated invasiveness of nasopharyngeal cancer cellsin vitro

(2009) Vivian Wai Yan Lui et al.   INTERNATIONAL JOURNAL OF CANCER

Epstein-Barr virus BART gene expression

(2009) M. Al-Mozaini et al.   JOURNAL OF GENERAL VIROLOGY

Inhibition of p38 MAP kinase pathway induces apoptosis and prevents Epstein Barr virus reactivation in Raji cells exposed to lytic cycle inducing compounds

(2009) Giulia Matusali et al.   Molecular Cancer

Recurrent chemical reactivations of EBV promotes genome instability and enhances tumor progression of nasopharyngeal carcinoma cells

(2008) Chih-Yeu Fang et al.   INTERNATIONAL JOURNAL OF CANCER

Inhibition of nasopharyngeal carcinoma growth by RTA-expressing baculovirus vectors containingoriP

(2008) Leyao Wang et al.   JOURNAL OF GENE MEDICINE