Development of gastric cancer in nonatrophic stomach with highly active inflammation identified by serum levels of pepsinogen andHelicobacter pyloriantibody together with endoscopic rugal hyperplastic gastritis

Inflammatory Processes Triggered by Helicobacter pylori Infection Cause Aberrant DNA Methylation in Gastric Epithelial Cells

(2010) T. Niwa et al.   CANCER RESEARCH

H. pylori-Eradication Therapy Increases RUNX3 Expression in the Glandular Epithelial Cells in Enlarged-Fold Gastritis

(2010) Masayuki Suzuki et al.   JOURNAL OF CLINICAL BIOCHEMISTRY AND NUTRITION

Stomach Carcinoma Incidence Patterns in the United States by Histologic Type and Anatomic Site

(2009) H. Wu et al.   CANCER EPIDEMIOLOGY BIOMARKERS & PREVENTION

Helicobacter pylori typing as a tool for tracking human migration

(2009) Y. Yamaoka   CLINICAL MICROBIOLOGY AND INFECTION

Eradication ofHelicobacter pyloriprevents cancer development in subjects with mild gastric atrophy identified by serum pepsinogen levels

(2009) Kimihiko Yanaoka et al.   INTERNATIONAL JOURNAL OF CANCER

Cancer High-Risk Subjects Identified by Serum Pepsinogen Tests: Outcomes after 10-Year Follow-up in Asymptomatic Middle-Aged Males

(2008) K. Yanaoka et al.   CANCER EPIDEMIOLOGY BIOMARKERS & PREVENTION

Risk of gastric cancer in asymptomatic, middle-aged Japanese subjects based on serum pepsinogen andHelicobacter pyloriantibody levels

(2008) Kimihiko Yanaoka et al.   INTERNATIONAL JOURNAL OF CANCER