MICL controls inflammation in rheumatoid arthritis

Clec12a Is an Inhibitory Receptor for Uric Acid Crystals that Regulates Inflammation in Response to Cell Death

(2014) Konstantin Neumann et al.   IMMUNITY

Genetics of rheumatoid arthritis contributes to biology and drug discovery

(2013) Yukinori Okada et al.   NATURE

Modulation of monosodium urate crystal-induced responses in neutrophils by the myeloid inhibitory C-type lectin-like receptor: potential therapeutic implications

(2013) Valérie Gagné et al.   ARTHRITIS RESEARCH & THERAPY

Signaling by Myeloid C-Type Lectin Receptors in Immunity and Homeostasis

(2012) David Sancho et al.   Annual Review of Immunology

Dectin-1 Is Not Required for Controlling Candida albicans Colonization of the Gastrointestinal Tract

(2012) Simon Vautier et al.   INFECTION AND IMMUNITY

A genetic association study of the CLEC12A gene in rheumatoid arthritis

(2012) Laëtitia Michou et al.   JOINT BONE SPINE

Induction of osteoclastogenesis and bone loss by human autoantibodies against citrullinated vimentin

(2012) Ulrike Harre et al.   JOURNAL OF CLINICAL INVESTIGATION

A Replication Study Confirms the Association of Dendritic Cell Immunoreceptor (DCIR) Polymorphisms with ACPA - Negative RA in a Large Asian Cohort

(2012) Jianping Guo et al.   PLoS One

The Pathogenesis of Rheumatoid Arthritis

(2011) Iain B. McInnes et al.   NEW ENGLAND JOURNAL OF MEDICINE

Inhibitory C-type lectin receptors in myeloid cells

(2010) Pierre Redelinghuys et al.   IMMUNOLOGY LETTERS

The C-Type Lectin Clec12A Present on Mouse and Human Dendritic Cells Can Serve as a Target for Antigen Delivery and Enhancement of Antibody Responses

(2009) M. H. Lahoud et al.   JOURNAL OF IMMUNOLOGY

Syk kinase is required for collaborative cytokine production induced through Dectin-1 and Toll-like receptors

(2008) Kevin M. Dennehy et al.   EUROPEAN JOURNAL OF IMMUNOLOGY

Characterisation of murine MICL (CLEC12A) and evidence for an endogenous ligand

(2008) Elwira Pyż et al.   EUROPEAN JOURNAL OF IMMUNOLOGY

Dcir deficiency causes development of autoimmune diseases in mice due to excess expansion of dendritic cells

(2008) Noriyuki Fujikado et al.   NATURE MEDICINE