Contribution of Bordetella Filamentous Hemagglutinin and Adenylate Cyclase Toxin to Suppression and Evasion of Interleukin-17-Mediated Inflammation
出版年份 2012 全文链接
标题
Contribution of Bordetella Filamentous Hemagglutinin and Adenylate Cyclase Toxin to Suppression and Evasion of Interleukin-17-Mediated Inflammation
作者
关键词
-
出版物
INFECTION AND IMMUNITY
Volume 80, Issue 6, Pages 2061-2075
出版商
American Society for Microbiology
发表日期
2012-04-03
DOI
10.1128/iai.00148-12
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- IL-17 and Therapeutic Kynurenines in Pathogenic Inflammation to Fungi
- (2014) L. Romani et al. JOURNAL OF IMMUNOLOGY
- Lipopolysaccharides from Bordetella pertussis and Bordetella parapertussis Differently Modulate Human Dendritic Cell Functions Resulting in Divergent Prevalence of Th17-Polarized Responses
- (2014) G. Fedele et al. JOURNAL OF IMMUNOLOGY
- A deep rough type structure in Bordetella bronchiseptica lipopolysaccharide modulates host immune responses
- (2011) Federico Sisti et al. MICROBIOLOGY AND IMMUNOLOGY
- Substitution of the Bordetella pertussis Lipid A Phosphate Groups with Glucosamine Is Required for Robust NF- B Activation and Release of Proinflammatory Cytokines in Cells Expressing Human but Not Murine Toll-Like Receptor 4-MD-2-CD14
- (2010) N. Marr et al. INFECTION AND IMMUNITY
- Pertactin Is Required for Bordetella Species To Resist Neutrophil-Mediated Clearance
- (2010) C. S. Inatsuka et al. INFECTION AND IMMUNITY
- Inflammasome Activation by Adenylate Cyclase Toxin Directs Th17 Responses and Protection against Bordetella pertussis
- (2010) A. Dunne et al. JOURNAL OF IMMUNOLOGY
- Candida albicans Dampens Host Defense by Downregulating IL-17 Production
- (2010) S.-C. Cheng et al. JOURNAL OF IMMUNOLOGY
- Bordetella pertussis Commits Human Dendritic Cells to Promote a Th1/Th17 Response through the Activity of Adenylate Cyclase Toxin and MAPK-Pathways
- (2010) Giorgio Fedele et al. PLoS One
- Development, regulation and functional capacities of Th17 cells
- (2010) Keiji Hirota et al. Seminars in Immunopathology
- Identification of a Novel Lipopolysaccharide Core Biosynthesis Gene Cluster in Bordetella pertussis, and Influence of Core Structure and Lipid A Glucosamine Substitution on Endotoxic Activity
- (2009) J. Geurtsen et al. INFECTION AND IMMUNITY
- IL-17 produced by neutrophils regulates IFN-γ–mediated neutrophil migration in mouse kidney ischemia-reperfusion injury
- (2009) Li Li et al. JOURNAL OF CLINICAL INVESTIGATION
- Natural-host animal models indicate functional interchangeability between the filamentous haemagglutinins ofBordetella pertussisandBordetella bronchisepticaand reveal a role for the mature C-terminal domain, but not the RGD motif, during infection
- (2009) Steven M. Julio et al. MOLECULAR MICROBIOLOGY
- Inefficient Toll-Like Receptor-4 Stimulation Enables Bordetella parapertussis to Avoid Host Immunity
- (2009) Daniel N. Wolfe et al. PLoS One
- Pertussis Toxin Stimulates IL-17 Production in Response to Bordetella pertussis Infection in Mice
- (2009) Charlotte Andreasen et al. PLoS One
- Interleukin-17 in host defence against bacterial, mycobacterial and fungal pathogens
- (2008) Meredith M. Curtis et al. IMMUNOLOGY
- MPO-ANCA induces IL-17 production by activated neutrophils in vitro via its Fc region- and complement-dependent manner
- (2008) Akiyoshi Hoshino et al. JOURNAL OF AUTOIMMUNITY
- Adenylate cycalse toxin of Bordetella pertussis inhibits TLR-induced IRF-1 and IRF-8 activation and IL-12 production and enhances IL-10 through MAPK activation in dendritic cells
- (2008) Fionnuala B. Hickey et al. JOURNAL OF LEUKOCYTE BIOLOGY
- Induction and effector functions of TH17 cells
- (2008) Estelle Bettelli et al. NATURE
- Modulation of the NF-κB Pathway by Bordetella pertussis Filamentous Hemagglutinin
- (2008) Tzvia Abramson et al. PLoS One
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExploreCreate your own webinar
Interested in hosting your own webinar? Check the schedule and propose your idea to the Peeref Content Team.
Create Now