标题
Age effects on mouse and human B cells
作者
关键词
Aging, B cells, Transcription factors, Antibody production, Vaccines
出版物
IMMUNOLOGIC RESEARCH
Volume 57, Issue 1-3, Pages 354-360
出版商
Springer Nature
发表日期
2013-11-07
DOI
10.1007/s12026-013-8440-9
参考文献
相关参考文献
注意：仅列出部分参考文献，下载原文获取全部文献信息。- Aging Down-Regulates the Transcription Factor E2A, Activation-Induced Cytidine Deaminase, and Ig Class Switch in Human B Cells
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- An age-related numerical and functional deficit in CD19+CD24hiCD38hiB cells is associated with an increase in systemic autoimmunity
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- A novel B cell population revealed by a CD38/CD24 gating strategy: CD38−CD24− B cells in centenarian offspring and elderly people
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- AID in aging and autoimmune diseases
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- Unique biomarkers for B-cell function predict the serum response to pandemic H1N1 influenza vaccine
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- Protein phosphatase 2A (PP2A) is increased in old murine B cells and mediates p38 MAPK/tristetraprolin dephosphorylation and E47 mRNA instability
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- Intrinsic defects in B cell response to seasonal influenza vaccination in elderly humans
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- Role of CMV in immune senescence
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- A double-negative (IgD−CD27−) B cell population is increased in the peripheral blood of elderly people
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- B cell–specific and stimulation-responsive enhancers derepress Aicda by overcoming the effects of silencers
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