Natriuretic Peptide Receptor-C Attenuates Hypertension in Spontaneously Hypertensive Rats

Sesamin ameliorates arterial dysfunction in spontaneously hypertensive rats via downregulation of NADPH oxidase subunits and upregulation of eNOS expression

(2013) Jun-xiu Zhang et al.   ACTA PHARMACOLOGICA SINICA

Effects of Allopurinol on Coronary Microvascular and Left Ventricular Function in Patients With Idiopathic Dilated Cardiomyopathy

(2012) Dogan Erdogan et al.   CANADIAN JOURNAL OF CARDIOLOGY

Whole-cell and nuclear NADPH oxidases levels and distribution in human endocardial endothelial, vascular smooth muscle, and vascular endothelial cells

(2012) Lena Ahmarani et al.   CANADIAN JOURNAL OF PHYSIOLOGY AND PHARMACOLOGY

A Blueberry-Enriched Diet Attenuates Nephropathy in a Rat Model of Hypertension via Reduction in Oxidative Stress

(2011) Carrie M. Elks et al.   PLoS One

Enhanced levels of endogenous endothelin-1 contribute to the over expression of Giα protein in vascular smooth muscle cells from SHR: Role of growth factor receptor activation

(2010) Yessica-Haydee Gomez Sandoval et al.   CELLULAR SIGNALLING

Stimulation of natriuretic peptide receptor C attenuates accumulation of reactive oxygen species and nitric oxide synthesis in ammonia-treated astrocytes

(2010) Marta Skowrońska et al.   JOURNAL OF NEUROCHEMISTRY

C-type natriuretic peptide effects on cardiovascular nitric oxide system in spontaneously hypertensive rats

(2010) Carolina Caniffi et al.   PEPTIDES

Nitric oxide attenuates the expression of natriuretic peptide receptor C and associated adenylyl cyclase signaling in aortic vascular smooth muscle cells: role of MAPK

(2009) Maria Arejian et al.   AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY

Natriuretic peptides stimulate the cardiac sodium pump via NPR-C-coupled NOS activation

(2008) M. William et al.   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

Activation of natriuretic peptide receptor-C attenuates the enhanced oxidative stress in vascular smooth muscle cells from spontaneously hypertensive rats: Implication of Giα protein

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