γδT Cell–Derived Interleukin-17A via an Interleukin-1β–Dependent Mechanism Mediates Cardiac Injury and Fibrosis in Hypertension

标题
γδT Cell–Derived Interleukin-17A via an Interleukin-1β–Dependent Mechanism Mediates Cardiac Injury and Fibrosis in Hypertension
作者
关键词
-
出版物
HYPERTENSION
Volume 64, Issue 2, Pages 305-314
出版商
Ovid Technologies (Wolters Kluwer Health)
发表日期
2014-05-28
DOI
10.1161/hypertensionaha.113.02604

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