Chronic pro-oxidative state and mitochondrial dysfunctions are more pronounced in fibroblasts from Down syndrome foeti with congenital heart defects

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Genetic analysis of Down syndrome-associated heart defects in mice

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The transcriptional coactivators, PGC-1α and β, cooperate to maintain cardiac mitochondrial function during the early stages of insulin resistance

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A forty-kilodalton protein of the inner membrane is the mitochondrial calcium uniporter

(2011) Diego De Stefani et al.   NATURE

Reactive Oxygen Species Production by Forward and Reverse Electron Fluxes in the Mitochondrial Respiratory Chain

(2011) Vitaly A. Selivanov et al.   PLoS Computational Biology

Oxidative stress caused by mitochondrial calcium overload

(2010) Tsung-I Peng et al.   Annals of the New York Academy of Sciences

Impairment of F1F0-ATPase, adenine nucleotide translocator and adenylate kinase causes mitochondrial energy deficit in human skin fibroblasts with chromosome 21 trisomy

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Metabolic control of mitochondrial biogenesis through the PGC-1 family regulatory network

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ChChd3, an Inner Mitochondrial Membrane Protein, Is Essential for Maintaining Crista Integrity and Mitochondrial Function

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Crosstalk signaling between mitochondrial Ca2+ and ROS

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SUMOylation Attenuates the Function of PGC-1α

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Effect of Coenzyme Q10 in mitigating oxidative DNA damage in Down syndrome patients, a double blind randomized controlled trial

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Elevated intracellular calcium in neutrophils in patients with Down syndrome

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Diphenyleneiodonium acutely inhibits reactive oxygen species production by mitochondrial complex I during reverse, but not forward electron transport

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Cristae formation—linking ultrastructure and function of mitochondria

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Oxidative stress during diabetic pregnancy disrupts cardiac neural crest migration and causes outflow tract defects

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Mitochondrial dysfunction in mouse trisomy 16 brain

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