Pnpla3I148Mknockin mice accumulate PNPLA3 on lipid droplets and develop hepatic steatosis
出版年份 2014 全文链接
标题
Pnpla3I148Mknockin mice accumulate PNPLA3 on lipid droplets and develop hepatic steatosis
作者
关键词
-
出版物
HEPATOLOGY
Volume 61, Issue 1, Pages 108-118
出版商
Wiley
发表日期
2014-06-10
DOI
10.1002/hep.27242
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Recombinant PNPLA3 protein shows triglyceride hydrolase activity and its I148M mutation results in loss of function
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- PNPLA3 I148M polymorphism and progressive liver disease
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- Chronic overexpression of PNPLA3I148M in mouse liver causes hepatic steatosis
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- Paradoxical Lower Serum Triglyceride Levels and Higher Type 2 Diabetes Mellitus Susceptibility in Obese Individuals with the PNPLA3 148M Variant
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- Expression and Characterization of a PNPLA3 Protein Isoform (I148M) Associated with Nonalcoholic Fatty Liver Disease
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- Genome-Wide Association Analysis Identifies Variants Associated with Nonalcoholic Fatty Liver Disease That Have Distinct Effects on Metabolic Traits
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- Increased hepatic fat in overweight Hispanic youth influenced by interaction between genetic variation in PNPLA3 and high dietary carbohydrate and sugar consumption
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- Patatin-like phospholipase domain-containing 3/adiponutrin deficiency in mice is not associated with fatty liver disease
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- A common variant in the patatin-like phospholipase 3 gene (PNPLA3) is associated with fatty liver disease in obese children and adolescents
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- PNPLA3variants specifically confer increased risk for histologic nonalcoholic fatty liver disease but not metabolic disease
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- Pnpla3/Adiponutrin deficiency in mice does not contribute to fatty liver disease or metabolic syndrome
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- Dissociation Between Fatty Liver and Insulin Resistance in Humans Carrying a Variant of the Patatin-Like Phospholipase 3 Gene
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- A common variant in PNPLA3, which encodes adiponutrin, is associated with liver fat content in humans
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- A Sequence Variation (I148M) in PNPLA3 Associated with Nonalcoholic Fatty Liver Disease Disrupts Triglyceride Hydrolysis
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- Deletion of ELOVL5 leads to fatty liver through activation of SREBP-1c in mice
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