Increased susceptibility of natural killer T-cell-deficient mice to acetaminophen-induced liver injury

Endogenous Interleukin-4 Regulates Glutathione Synthesis Following Acetaminophen-Induced Liver Injury in Mice

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Alternatively activated macrophages produce catecholamines to sustain adaptive thermogenesis

(2011) Khoa D. Nguyen et al.   NATURE

Impact of CD1d Deficiency on Metabolism

(2011) Maya E. Kotas et al.   PLoS One

Eosinophils Sustain Adipose Alternatively Activated Macrophages Associated with Glucose Homeostasis

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Primary deficiency of microsomal triglyceride transfer protein in human abetalipoproteinemia is associated with loss of CD1 function

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Absence of invariant natural killer T cells deteriorates liver inflammation and fibrosis in mice fed high-fat diet

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Hyperketonemia decreases mitochondrial membrane potential and its normalization with chromium (III) supplementation in monocytes

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IL-4/STAT6 immune axis regulates peripheral nutrient metabolism and insulin sensitivity

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Role of oxidative stress in alcohol-induced liver injury

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Pathogenic role of natural killer T and natural killer cells in acetaminophen-induced liver injury in mice is dependent on the presence of dimethyl sulfoxide

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Role of JNK Translocation to Mitochondria Leading to Inhibition of Mitochondria Bioenergetics in Acetaminophen-induced Liver Injury

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