Oxidative stress, Nrf2 and keratin up-regulation associate with Mallory-Denk body formation in mouse erythropoietic protoporphyria
出版年份 2012 全文链接
标题
Oxidative stress, Nrf2 and keratin up-regulation associate with Mallory-Denk body formation in mouse erythropoietic protoporphyria
作者
关键词
-
出版物
HEPATOLOGY
Volume 56, Issue 1, Pages 322-331
出版商
Wiley
发表日期
2012-02-15
DOI
10.1002/hep.25664
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Mallory–Denk Bodies Are Associated With Outcomes and Histologic Features in Patients With Chronic Hepatitis C
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- Protoporphyrin Retention in Hepatocytes and Kupffer Cells Prevents Sclerosing Cholangitis in Erythropoietic Protoporphyria Mouse Model
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- Energy determinants GAPDH and NDPK act as genetic modifiers for hepatocyte inclusion formation
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- Persistent activation of Nrf2 through p62 in hepatocellular carcinoma cells
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- Porphyrias
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- Keratins modulate the shape and function of hepatocyte mitochondria: a mechanism for protection from apoptosis
- (2009) G.-Z. Tao et al. JOURNAL OF CELL SCIENCE
- Influence of ethnicity on histological differences in non-alcoholic fatty liver disease
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- A Novel Role of the NRF2 Transcription Factor in the Regulation of Arsenite-Mediated Keratin 16 Gene Expression in Human Keratinocytes
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- The genetic background modulates susceptibility to mouse liver Mallory-Denk body formation and liver injury
- (2008) Shinichiro Hanada et al. HEPATOLOGY
- Autophagy activation by rapamycin eliminates mouse Mallory-Denk bodies and blocks their proteasome inhibitor-mediated formation
- (2008) Masaru Harada et al. HEPATOLOGY
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